Common questions

What causes late onset ataxia?

What causes late onset ataxia?

Persistent ataxia usually results from damage to the part of your brain that controls muscle coordination (cerebellum). Many conditions can cause ataxia, including alcohol misuse, certain medication, stroke, tumor, cerebral palsy, brain degeneration and multiple sclerosis.

How long can you live with spinocerebellar ataxia?

Signs and symptoms of the disorder typically begin in early adulthood but can appear anytime from childhood to late adulthood. People with SCA1 typically survive 10 to 20 years after symptoms first appear.

When should you suspect spinocerebellar ataxia?

A diagnosis of spinocerebellar ataxia (SCA) is often suspected when certain signs and symptoms, such as a poorly coordinated gait (walk) and uncoordinated hand/finger movements, are present.

Will an MRI show ataxia?

Imaging studies. A CT scan or MRI of your brain might help determine potential causes. An MRI can sometimes show shrinkage of the cerebellum and other brain structures in people with ataxia. It may also show other treatable findings, such as a blood clot or benign tumor, that could be pressing on your cerebellum.

Does ataxia affect breathing?

The weakness of these muscles provokes in an ataxic person a respiratory difficulty which manifests itself by breathlessness, even for moderate work. Ataxic persons show difficulty to accomplish several actions while maintaining their breathing. To converse, to walk and to breathe simultaneously becomes difficult.

Is ataxia a symptom of ALS?

In the most severe cases, ataxia resembles ALS, making it difficult to swallow and eventually to breathe. “At this point there’s nothing we can do to slow the pace of their disease.”

Does anyone know possible ataxia symptoms?

What are the symptoms of ataxia? Some of the most common symptoms of ataxia can include: problems with coordination and balance, which can include clumsiness , an unsteady gait, and frequent falling

Is there a cure for Ataxia telangiectasia?

There is no cure for A-T at this time. The cloning and sequencing of the gene (named ATM, for ataxia telangiectasia, mutated) has opened several avenues of research to develop better treatment, including: Gene therapy The design of drugs to correct the function of the altered protein Direct replacement of the functional protein.